[1] Kikuta J, Yamaguchi M, Shimizu M,et al. Notch signaling induces root resorption via RANKL and IL-6 from hPDL cells. J Dent Res. 2015;94(1):140-147.[2] Chang L, Feng X, Gao W. Proliferation of rheumatoid arthritis fibroblast-like synoviocytes is enhanced by IL-17-mediated autophagy through STAT3 activation. Connective tissue research. Connect Tissue Res. 2018 Nov 26. .[3] Batool H, Nadeem A, Kashif M, et al. Salivary Levels of IL-6 and IL-17 Could Be an Indicator of Disease Severity in Patients with Calculus Associated Chronic Periodontitis. BioMed research international. Biomed Res Int. 2018;2018:8531961. [4] Wang ZX, Yang L, Tan JY, et al. [Effects of T helper 1 cells and T helper 17 cells secreting cytokines on rat models of experimental periodontitis]. Zhonghua kou qiang yi xue za zhi.2017;52(12):740-747.[5] Meikle MC. The tissue, cellular, and molecular regulation of orthodontic tooth movement: 100 years after Carl Sandstedt. Eur J Orthod. 2006;28(3):221-240.[6] Lee YH, Nahm DS, Jung YK, et al. Differential gene expression of periodontal ligament cells after loading of static compressive force. J Periodontol. 2007;78(3):446-452.[7] Schindler JF, Monahan JB, Smith WG. p38 pathway kinases as anti-inflammatory drug targets. J Dent Res. 2007;86(9): 800-811. [8] 张宝平. 癌细胞力学性质测量及放射诱导下细胞损伤的生物力学研究[D]: 兰州:兰州大学,2016.[9] 张西正,匡震邦,蔡绍皙,等. 细胞力学实验技术研究[J]. 实验力学,2001,16(1):66-76.[10] Redlich M, Roos H, Reichenberg E, et al. The effect of centrifugal force on mRNA levels of collagenase, collagen type-I, tissue inhibitors of metalloproteinases and beta-actin in cultured human periodontal ligament fibroblasts. J Periodontal Res. 2004;39(1):27-32.[11] Yousefian J, Firouzian F, Shanfeld J, et al. A new experimental model for studying the response of periodontal ligament cells to hydrostatic pressure. Am J Orthod Dentofacial Orthop. 1995;108(4):402-409..[12] 朱庆党,巢永烈,陈新民,等. 动态张、压应力刺激下人牙周膜成纤维细胞细胞骨架变化[J]. 实用口腔医学杂志. 2008,24(4): 559-562.[13] Kanzaki H,Chiba M,Shimizu Y,et al.Periodontal ligament cells under mechanical stress induce osteoclastogenesis by receptor activator of nuclear factor kappaB ligand up-regulation via prostaglandin E2 synthesis. J Bone Miner Res. 2002;17(2):210-220.[14] 张建兴,黄生高,钟孝欢,等. 人牙周膜细胞体外培养和机械压力模型构建[J].口腔医学研究,2006,22(1):38-42.[15] Yamaguchi M, Aihara N, Kojima T, et al. RANKL increase in compressed periodontal ligament cells from root resorption. J Dent Res. 2006;85(8):751-756.[16] Cargnello M, Roux PP. Activation and function of the MAPKs and their substrates, the MAPK-activated protein kinases. Microbiology and molecular biology reviews : MMBR. 2011; 75(1):50-83.[17] Ouyang W,Kolls JK,Zheng Y.The biological functions of T helper 17 cell effector cytokines in inflammation. Immunity. 2008;28(4):454-67.[18] Xing J,Yu Z,Zhang X, et al. Epicatechin alleviates inflammation in lipopolysaccharide-induced acute lung injury in mice by inhibiting the p38 MAPK signaling pathway. Int Immunopharmacol. 2018;66:146-153.[19] Ziegler N, Alonso A, Steinberg T, et al. Mechano-transduction in periodontal ligament cells identifies activated states of MAP-kinases p42/44 and p38-stress kinase as a mechanism for MMP-13 expression. BMC cell biology. 2010;11:10.[20] 李菲菲,阎潇,王庆,等. p38丝裂素活化蛋白激酶信号通路在应力调控成肌细胞分化中的作用.[J] 华西口腔医学杂志, 2012;30(6): 574-578.[21] Yang JZ, Li Y, Sun LX, et al. Interleukin-17 receptor expression on vascular endothelial cells of masses of skeletal extramedullary disease in myeloma patients. Pathol Res Pract. 2014;210(9):586-590..[22] Hayashi N, Yamaguchi M, Nakajima R,et al. T-helper 17 cells mediate the osteo/odontoclastogenesis induced by excessive orthodontic forces. Oral Dis. 2012;18(4):375-388[23] Funaki Y, Hasegawa Y, Okazaki R, et al. Resolvin E1 Inhibits Osteoclastogenesis and Bone Resorption by Suppressing IL-17-induced RANKL Expression in Osteoblasts and RANKL-induced Osteoclast Differentiation. Yonago Acta Med. 2018;61(1):8-18.[24] Yamada K, Yamaguchi M, Asano M, et al. Th17-cells in atopic dermatitis stimulate orthodontic root resorption. Oral Dis. 2013;19(7):683-693.[25] Liu Y, Song F, Wu S, et al. Protein and mRNA expressions of IL-6 and its key signaling factors under orthodontic forces in mice: An in-vivo study.Am J Orthod Dentofacial Orthop. 2017; 152(5):654-662..[26] Koyama Y, Mitsui N, Suzuki N, et al. Effect of compressive force on the expression of inflammatory cytokines and their receptors in osteoblastic Saos-2 cells. Arch Oral Biol. 2008 ; 53(5):488-496. [27] Ganesan R,Rasool M.Interleukin 17 regulates SHP-2 and IL-17RA/STAT-3 dependent Cyr61, IL-23 and GM-CSF expression and RANKL mediated osteoclastogenesis by fibroblast-like synoviocytes in rheumatoid arthritis. Mol Immunol. 2017;91:134-144 .[28] Johnson RW,McGregor NE,Brennan HJ,et al.Glycoprotein130 (Gp130)/interleukin-6 (IL-6) signalling in osteoclasts promotes bone formation in periosteal and trabecular bone. Bone. 2015; 81:343-351.[29] 余学清,李锦华,黄凌虹,等. p38信号通路在脂多糖诱导的大鼠系膜细胞产生白细胞介素1β中的作用[J].中华肾脏病杂志. 2000, 16(2):98-101. |