[1] WEE NK, KULKARNI RN, HORSNELL H, et al. The brain in bone and fuel metabolism. Bone. 2016;82:56-63.
[2] SUN P, HE L, JIA K, et al. Regulation of body length and bone mass by Gpr126/Adgrg6. Sci Adv. 2020;6(12):eaaz0368.
[3] 刘媛.骨源性LCN2在不同方式运动调控骨—下丘脑轴中的作用和机制[D].上海:华东师范大学,2019.
[4] MASI L. Crosstalk between the brain and bone. Clin Cases Miner Bone Metab. 2012;9(1):13-16.
[5] 徐帅,陈祥和,李世昌.下丘脑介导能量代谢与骨骼反调节及其运动干预机制研究进展[J].中国体育科技,2021,57(2):74-81.
[6] IDELEVICH A, BARON R. Brain to bone: What is the contribution of the brain to skeletal homeostasis? Bone. 2018;115:31-42.
[7] 于小奎,朱兵.神经肽Y对骨代谢调节的研究进展[J].中国骨质疏松杂志, 2013,19(12):1323-1326.
[8] LEE NJ, QI Y, ENRIQUEZ RF, et al. Lack of NPY in neurotensin neurons leads to a lean phenotype. Neuropeptides. 2020;80:101994.
[9] BALDOCK PA, ALLISON SJ, LUNDBERG P, et al. Novel role of Y1 receptors in the coordinated regulation of bone and energy homeostasis. J Biol Chem. 2007; 282(26):19092-19102.
[10] SOUSA DM, CONCEIÇÃO F, SILVA DI, et al. Ablation of Y1 receptor impairs osteoclast bone-resorbing activity. Sci Rep. 2016;6:33470.
[11] YAO D, HUANG L, KE J, et al. Bone metabolism regulation: Implications for the treatment of bone diseases. Biomed Pharmacother. 2020;129:110494.
[12] SHI YC, BALDOCK PA. Central and peripheral mechanisms of the NPY system in the regulation of bone and adipose tissue. Bone. 2012;50(2):430-436.
[13] CATAK Z, AYDIN S, SAHIN I, et al. Regulatory neuropeptides (ghrelin, obestatin and nesfatin-1) levels in serum and reproductive tissues of female and male rats with fructose-induced metabolic syndrome. Neuropeptides. 2014;48(3):167-177.
[14] ELEFTERIOU F. Regulation of bone remodeling by the central and peripheral nervous system. Arch Biochem Biophys. 2008;473(2):231-236.
[15] SELDEEN KL, HALLEY PG, VOLMAR CH, et al. Neuropeptide Y Y2 antagonist treated ovariectomized mice exhibit greater bone mineral density. Neuropeptides. 2018; 67:45-55.
[16] 徐帅,李世昌,孙朋.神经肽Y介导的骨代谢及运动干预调控研究[J].中华骨质疏松和骨矿盐疾病杂志,2018,11(2):193-199.
[17] LUNDBERG P, ALLISON SJ, LEE NJ, et al. Greater bone formation of Y2 knockout mice is associated with increased osteoprogenitor numbers and altered Y1 receptor expression. J Biol Chem. 2007;282(26):19082-19091.
[18] BALDOCK PA, SAINSBURY A, COUZENS M, et al. Hypothalamic Y2 receptors regulate bone formation. J Clin Invest. 2002;109(7):915-921.
[19] QI Y, FU M, HERZOG H. Y2 receptor signalling in NPY neurons controls bone formation and fasting induced feeding but not spontaneous feeding. Neuropeptides. 2016;55:91-97.
[20] UPADHYAY J, FARR OM, MANTZOROS CS. The role of leptin in regulating bone metabolism. Metabolism. 2015;64(1):105-113.
[21] SHAO P, WANG Y, ZHANG M, et al. The interference of DEHP in precocious puberty of females mediated by the hypothalamic IGF-1/PI3K/Akt/mTOR signaling pathway. Ecotoxicol Environ Saf. 2019;181:362-369.
[22] FERNÁNDEZ-GALAZ MC, FERNÁNDEZ-AGULLÓ T, CARRASCOSA JM, et al. Leptin accumulation in hypothalamic and dorsal raphe neurons is inversely correlated with brain serotonin content. Brain Res. 2010;1329:194-202.
[23] MANTZOROS CS, MAGKOS F, BRINKOETTER M, et al. Leptin in human physiology and pathophysiology. Am J Physiol Endocrinol Metab. 2011;301(4):E567-E584.
[24] LEE NJ, CLARKE IM, ENRIQUEZ RF, et al. Central RANK signalling in NPY neurons alters bone mass in male mice. Neuropeptides. 2018;68:75-83.
[25] FRANCISCO V, PÉREZ T, PINO J, et al. Biomechanics, obesity, and osteoarthritis. The role of adipokines: When the levee breaks. J Orthop Res. 2018;36(2):594-604.
[26] MENG XH, TAN LJ, XIAO HM, et al. Examining the causal role of leptin in bone mineral density: A Mendelian randomization study. Bone. 2019;125:25-29.
[27] DIMITRI P, ROSEN C. The Central Nervous System and Bone Metabolism: An Evolving Story. Calcif Tissue Int. 2017;100(5):476-485.
[28] SINGH S, SINGH S. JAK-STAT inhibitors: Immersing therapeutic approach for management of rheumatoid arthritis. Int Immunopharmacol. 2020;86:106731.
[29] CHOPIN F, BIVER E, FUNCK-BRENTANO T, et al. Prognostic interest of bone turnover markers in the management of postmenopausal osteoporosis. Joint Bone Spine. 2012;79(1):26-31.
[30] SAHIN ERSOY G, GIRAY B, SUBAS S, et al. Interpregnancy interval as a risk factor for postmenopausal osteoporosis. Maturitas. 2015;82(2):236-240.
[31] 董万涛,吕泽斌,宋敏,等.从脾肾论治骨质疏松的神经-内分泌-免疫网络平衡机制[J].中国骨质疏松杂志,2015,21(11):1416-1419.
[32] EL HADIDY EL HM, GHONAIM M, El Gawad SSh, et al. Impact of severity, duration, and etiology of hyperthyroidism on bone turnover markers and bone mineral density in men. BMC Endocr Disord. 2011;11:15.
[33] MOSIALOU I, SHIKHEL S, LIU JM, et al. Corrigendum: MC4R-dependent suppression of appetite by bone-derived lipocalin 2. Nature. 2017;546(7658):440.
[34] 董万涛,周灵通,宋敏,等.固本增骨方对去卵巢大鼠血清骨钙素和NEI网络组织中游离[Ca2+]i的影响[J].中药药理与临床,2018,34(1):121-127.
[35] GUEDES JAC, ESTEVES JV, MORAIS MR, et al. Osteocalcin improves insulin resistance and inflammation in obese mice: Participation of white adipose tissue and bone. Bone. 2018;115:68-82.
[36] KONDO A, TOKUDA H, KATO K, et al. Rho-kinase negatively regulates thyroid hormone-stimulated osteocalcin synthesis in osteoblasts. Biochimie. 2013;95(4): 719-724.
[37] 苏俊平,张博,陈云霞,等.甲状腺功能亢进者治疗前后甲状腺激素和血清骨钙素的变化[J].宁夏医科大学学报,2017,39(3):303-305.
[38] MIZOKAMI A, KAWAKUBO-YASUKOCHI T, HIRATA M. Osteocalcin and its endocrine functions. Biochem Pharmacol. 2017;132:1-8.
[39] CHOU SH, MANTZOROS C. Bone metabolism in anorexia nervosa and hypothalamic amenorrhea. Metabolism. 2018;80:91-104.
[40] YAO Q, LIANG H, HUANG B, et al. Beta-adrenergic signaling affect osteoclastogenesis via osteocytic MLO-Y4 cells’ RANKL production. Biochem Biophys Res Commun. 2017;488(4):634-640.
[41] MANGLANI K, VIJAYAN V, PATHAK C, et al. Development and characterization of supramolecular calcitonin assembly and assessment of its interactions with the bone remodelling process. Bone. 2019;122:123-135.
[42] ENDO N, FUJINO K, DOI T, et al. Effect of elcatonin versus nonsteroidal anti-inflammatory medications for acute back pain in patients with osteoporotic vertebral fracture: a multiclinic randomized controlled trial. J Bone Miner Metab. 2017;35(4):375-384.
[43] YAZDANI J, KHIAVI RK, GHAVIMI MA, et al. Calcitonin as an analgesic agent: review of mechanisms of action and clinical applications. Braz J Anesthesiol. 2019;69(6): 594-604.
[44] 杨念恩.不同方式运动对生长期小鼠骨合成代谢和Wnt信号通路的影响[D].上海:华东师范大学,2014.
[45] 高飞,周武,谢卯,等.PFNA联合规范化抗骨质疏松药物治疗老年骨质疏松性股骨转子间骨折[J].第二军医大学学报,2017,38(4):437-442.
[46] 刘锡仪,刘浩宇.下丘脑弓状核在骨质疏松发病机制中的作用[J].广东医学院学报,2014,32(6):881-885.
[47] 孙晓琪.雌二醇通过G蛋白偶联雌激素受体30(GPR30)/ERK1/2信号通路调节MC3T3-E1细胞线粒体自噬的分子机制研究[D].沈阳:中国医科大学,2018.
[48] 李微,张博,张雨薇,等.雌激素调节骨代谢作用的研究进展[J].中国骨质疏松杂志,2017,23(2):262-266.
[49] 范金柱,杨柳,罗卓荆,等.雌激素对绝经后骨质疏松患者骨髓间充质干细胞Notch信号通路的影响[J].中华骨质疏松和骨矿盐疾病杂志,2013,6(3):232-239.
[50] 张萌萌.雌激素与雌激素受体骨代谢调节作用[J].中国骨质疏松杂志,2019, 25(5):704-708.
[51] KLEIN-NULEND J, VAN OERS RF, BAKKER AD, et al. Bone cell mechanosensitivity, estrogen deficiency, and osteoporosis. J Biomech. 2015;48(5):855-865.
[52] YUAN Y, CHEN X, ZHANG L, et al. The roles of exercise in bone remodeling and in prevention and treatment of osteoporosis. Prog Biophys Mol Biol. 2016;122(2): 122-130.
[53] HUHMANN K. Menses Requires Energy: A Review of How Disordered Eating, Excessive Exercise, and High Stress Lead to Menstrual Irregularities. Clin Ther. 2020;42(3):401-407.
[54] 秦晋泽,荣晓旭,朱国兴,等.广场舞对绝经期后骨质疏松患者的骨密度和骨转换指标影响的研究[J].中国骨质疏松杂志,2017,23(1):43-46,50.
[55] GOLDBER GA. Postm enopausal obesity risk a reduced when aerobic exercise is added to weight loss. Women Health Weakly. 2002;19(2):20.
[56] PEREIRA LJ, MACARI S, COIMBRA CC, et al. Aerobic and resistance training improve alveolar bone quality and interferes with bone-remodeling during orthodontic tooth movement in mice. Bone. 2020;138:115496.
[57] 陈鑫林.运动对原发性骨质疏松的干预与机制[J].中国组织工程研究,2018, 22(8):1294-1299.
[58] 徐瑞,严翊,谢敏豪.性激素反馈调节下丘脑ARC和AVPV中kisspeptin-GPR54信号通路的核团差异[J].中国生物化学与分子生物学报,2018,34(8): 810-817.
[59] 李良,徐建方,房国梁,等.有氧运动或抗阻运动诱导骨形态发生蛋白7调节大鼠能量代谢的研究[J].中国运动医学杂志,2019,38(11):960-968.
[60] 龚文辉,张素梅,储珏.有氧运动对饮食诱导的肥胖鼠下丘脑和褐色脂肪BMP7表达的影响[J].中国康复,2016,31(2):114-117.
[61] CHEN C, BAI GC, JIN HL, et al. Local injection of bone morphogenetic protein 7 promotes neuronal regeneration and motor function recovery after acute spinal cord injury. Neural Regen Res. 2018;13(6):1054-1060.
[62] REID IR. Short-term and long-term effects of osteoporosis therapies. Nat Rev Endocrinol. 2015;11(7):418-428.
[63] 王璐,陈媛媛,阮彩莲.有氧运动对焦虑大鼠血清促肾上腺皮质激素、三碘甲状腺原氨酸、甲状腺素及促甲状腺激素的影响[J].中国老年学杂志,2015, 35(8):2174-2175.
[64] 朱于青,孙琳.促甲状腺激素与骨代谢[J].中国医师杂志,2019,21(2):315-318.
[65] PRIDEAUX M, KITASE Y, KIMBLE M, et al. Taurine, an osteocyte metabolite, protects against oxidative stress-induced cell death and decreases inhibitors of the Wnt/β-catenin signaling pathway. Bone. 2020;137:115374.
[66] 杨念恩,李世昌.降钙素在不同方式运动影响骨形成代谢过程中的机制研究[A]//中国体育科学学会运动生理与生物化学分会.第四届(2016)全国运动生理与生物化学学术会议论文集[C].无锡:第四届(2016)全国运动生理与生物化学学术会议,2016:69.
[67] GRIMSTON SK, TANGUAY KE, GUNDBERG CM, et al. The calciotropic hormone response to changes in serum calcium during exercise in female long distance runners. J Clin Endocrinol Metab. 1993;76(4):867-872.
[68] POLITO R, MONDA V, NIGRO E, et al. The Important Role of Adiponectin and Orexin-A, Two Key Proteins Improving Healthy Status: Focus on Physical Activity. Front Physiol. 2020;11:356.
[69] ZHANG Y, KUMAGAI K, SAITO T. Effect of parathyroid hormone on early chondrogenic differentiation from mesenchymal stem cells. J Orthop Surg Res. 2014;9:68.
[70] O’ BRIEN MH, DUTRA EH, LIMA A, et al. PTH [1-34] induced differentiation and mineralization of mandibular condylar cartilage. Sci Rep. 2017;7(1):3226.
[71] 赵丹,施丹,史晓.围绝经期女性预防骨质疏松症研究进展[J].辽宁中医药大学学报,2019,21(2):118-121.
[72] LIU SS, ZHOU P, ZHANG Y. Abnormal expression of key genes and proteins in the canonical Wnt/β-catenin pathway of articular cartilage in a rat model of exercise-induced osteoarthritis. Mol Med Rep. 2016;13(3):1999-2006.
[73] CHENG L, KHALAF AT, LIN T, et al. Exercise Promotes the Osteoinduction of HA/β-TCP Biomaterials via the Wnt Signaling Pathway. Metabolites. 2020;10(3):90.
|