[1] DIKIC I, ELAZAR Z. Mechanism and medical implications of mammalian autophagy. Nat Rev Mol Cell Biol. 2018;19(6):349-364.
[2] YIN X, ZHOU C, LI J, et al. Autophagy in bone homeostasis and the onset of osteoporosis. Bone Res. 2019;7:28.
[3] RUBINSZTEIN DC, MARIñO G, KROEMER G. Autophagy and aging. Cell. 2011;146(5):682-695.
[4] TAN YQ, ZHANG J, ZHOU G. Autophagy and its implication in human oral diseases. Autophagy. 2017;13(2):225-236.
[5] CHEN X, WANG Z, DUAN N, et al. Osteoblast-osteoclast interactions. Connect Tissue Res. 2018;59(2):99-107.
[6] 孙友强,马超,梁萌梦,等.自噬在骨细胞中重要作用的最新研究进展:骨相关细胞活性和骨代谢[J].中国组织工程研究,2022, 26(2):276-282.
[7] WANG L, HAUENSTEIN AV. The NLRP3 inflammasome: Mechanism of action, role in disease and therapies. Mol Aspects Med. 2020;76: 100889.
[8] LI Y, LING J, JIANG Q. Inflammasomes in Alveolar Bone Loss. Front Immunol. 2021;12:691013.
[9] FLORENCIO-SILVA R, SASSO GR, SIMÕES MJ, et al. Osteoporosis and autophagy: What is the relationship? Rev Assoc Med Bras (1992). 2017; 63(2):173-179.
[10] HUANG L, JIN P, LIN X, et al. Beneficial effects of sulfonamide‑based gallates on osteoblasts in vitro. Mol Med Rep. 2017;15(3):1149-1156.
[11] NOLLET M, SANTUCCI-DARMANIN S, BREUIL V, et al. Autophagy in osteoblasts is involved in mineralization and bone homeostasis. Autophagy. 2014;10(11):1965-1977.
[12] 连璐雅,朱海华.自噬对炎症性牙槽骨吸收作用的研究进展[J].中国临床新医学,2020,13(8):833-836.
[13] LIU W, ZHAO Z, NA Y, et al. Dexamethasone-induced production of reactive oxygen species promotes apoptosis via endoplasmic reticulum stress and autophagy in MC3T3-E1 cells. Int J Mol Med. 2018;41(4): 2028-2036.
[14] ZHANG S, LIU Y, LIANG Q. Low-dose dexamethasone affects osteoblast viability by inducing autophagy via intracellular ROS. Mol Med Rep. 2018;17(3):4307-4316.
[15] WANG L, ZHANG H, DONG M, et al. Role of the Btk-PLCγ2 Signaling Pathway in the Bone Destruction of Apical Periodontitis. Mediators Inflamm. 2019;2019:8767529.
[16] SONG L, TAN J, WANG Z, et al. Interleukin‑17A facilitates osteoclast differentiation and bone resorption via activation of autophagy in mouse bone marrow macrophages. Mol Med Rep. 2019;19(6):4743-4752.
[17] 杨彩会,刘启成,董明,等.丝氨酸/苏氨酸蛋白激酶促进慢性根尖周炎模型小鼠的骨破坏[J].中国组织工程研究,2021,25(23): 3654-3659.
[18] LI RF, CHEN G, REN JG, et al. The adaptor protein p62 is involved in RANKL-induced autophagy and osteoclastogenesis. J Histochem Cytochem. 2014;62(12):879-888.
[19] SUL OJ, PARK HJ, SON HJ, et al. Lipopolysaccharide (LPS)-Induced Autophagy Is Responsible for Enhanced Osteoclastogenesis. Mol Cells. 2017;40(11):880-887.
[20] DESELM CJ, MILLER BC, ZOU W, et al. Autophagy proteins regulate the secretory component of osteoclastic bone resorption. Dev Cell. 2011;21(5):966-974.
[21] LIN NY, CHEN CW, KAGWIRIA R, et al. Inactivation of autophagy ameliorates glucocorticoid-induced and ovariectomy-induced bone loss. Ann Rheum Dis. 2016;75(6):1203-1210.
[22] 杨钰粒,徐亦文,安蒂,等.自噬在破骨细胞中的调控作用[J].生命的化学,2016,36(3):354-358.
[23] SCHRODER K, TSCHOPP J. The inflammasomes. Cell. 2010;140(6):821-832.
[24] CHEN Y, YANG Q, LV C, et al. NLRP3 regulates alveolar bone loss in ligature-induced periodontitis by promoting osteoclastic differentiation. Cell Prolif. 2021;54(2):e12973.
[25] SAITOH T, FUJITA N, JANG MH, et al. Loss of the autophagy protein Atg16L1 enhances endotoxin-induced IL-1beta production. Nature. 2008;456(7219):264-268.
[26] LEVINE B, MIZUSHIMA N, VIRGIN HW. Autophagy in immunity and inflammation. Nature. 2011;469(7330):323-335.
[27] BIASIZZO M, KOPITAR-JERALA N. Interplay Between NLRP3 Inflammasome and Autophagy. Front Immunol. 2020;11:591803.
[28] NETEA-MAIER RT, PLANTINGA TS, VAN DE VEERDONK FL, et al. Modulation of inflammation by autophagy: Consequences for human disease. Autophagy. 2016;12(2):245-260.
[29] ZHONG Z, SANCHEZ-LOPEZ E, KARIN M. Autophagy, NLRP3 inflammasome and auto-inflammatory/immune diseases. Clin Exp Rheumatol. 2016;34(4 Suppl 98):12-16.
[30] CAO Z, WANG Y, LONG Z, et al. Interaction between autophagy and the NLRP3 inflammasome. Acta Biochim Biophys Sin (Shanghai). 2019; 51(11):1087-1095.
[31] COSIN-ROGER J, SIMMEN S, MELHEM H, et al. Hypoxia ameliorates intestinal inflammation through NLRP3/mTOR downregulation and autophagy activation. Nat Commun. 2017;8(1):98.
[32] LI H, MIAO W, MA J, et al. Acute Exercise-Induced Mitochondrial Stress Triggers an Inflammatory Response in the Myocardium via NLRP3 Inflammasome Activation with Mitophagy. Oxid Med Cell Longev. 2016; 2016:1987149.
[33] ZOU YJ, XU JJ, WANG X, et al. Lactobacillus johnsonii L531 Ameliorates Escherichia coli-Induced Cell Damage via Inhibiting NLRP3 Inflammasome Activity and Promoting ATG5/ATG16L1-Mediated Autophagy in Porcine Mammary Epithelial Cells. Vet Sci. 2020;7(3):112.
[34] DUPONT N, JIANG S, PILLI M, et al. Autophagy-based unconventional secretory pathway for extracellular delivery of IL-1β. EMBO J. 2011; 30(23):4701-4711.
[35] AHMED ME, IYER S, THANGAVEL R, et al. Co-Localization of Glia Maturation Factor with NLRP3 Inflammasome and Autophagosome Markers in Human Alzheimer’s Disease Brain. J Alzheimers Dis. 2017; 60(3):1143-1160.
[36] ZHANG J, HUANG X, LU B, et al. Can apical periodontitis affect serum levels of CRP, IL-2, and IL-6 as well as induce pathological changes in remote organs. Clin Oral Investig. 2016;20(7):1617-1624.
[37] JAKOVLJEVIC A, MILETIC M, NIKOLIC N, et al. Notch signaling pathway mediates alveolar bone resorption in apical periodontitis. Med Hypotheses. 2019;124:87-90.
[38] WANG L, JIN H, YE D, et al. Enterococcus faecalis Lipoteichoic Acid-induced NLRP3 Inflammasome via the Activation of the Nuclear Factor Kappa B Pathway. J Endod. 2016;42(7):1093-1100.
[38] SHI CS, SHENDEROV K, HUANG NN, et al. Activation of autophagy by inflammatory signals limits IL-1β production by targeting ubiquitinated inflammasomes for destruction. Nat Immunol. 2012;13(3):255-263.
[40] PARK MH, JEONG SY, NA HS, et al. Porphyromonas gingivalis induces autophagy in THP-1-derived macrophages. Mol Oral Microbiol. 2017; 32(1):48-59.
[41] LAI EH, HONG CY, KOK SH, et al. Simvastatin alleviates the progression of periapical lesions by modulating autophagy and apoptosis in osteoblasts. J Endod. 2012;38(6):757-763.
[42] ZHU L, YANG J, ZHANG J, et al. The presence of autophagy in human periapical lesions. J Endod. 2013;39(11):1379-1384.
[43] HUANG HY, WANG WC, LIN PY, et al. The roles of autophagy and hypoxia in human inflammatory periapical lesions. Int Endod J. 2018; 51 Suppl 2:e125-e145.
[44] 马金宝,王倩,董明,等.人牙髓炎及根尖周炎病损组织中自噬相关蛋白微管相关蛋白1轻链3B的表达[J].中国组织工程研究,2019, 23(15):2364-2368.
[45] ALVAREZ VE, KOSEC G, SANT’ANNA C, et al. Autophagy is involved in nutritional stress response and differentiation in Trypanosoma cruzi. J Biol Chem. 2008;283(6):3454-3464.
[46] MYSAK J, PODZIMEK S, SOMMEROVA P, et al. Porphyromonas gingivalis: major periodontopathic pathogen overview. J Immunol Res. 2014; 2014:476068.
[47] 赵妍,于阳,寇育荣.牙龈卟啉单胞菌调控细胞自噬的分子机制研究进展[J].华西口腔医学杂志,2017,35(6):654-658.
[48] LEE K, ROBERTS JS, CHOI CH, et al. Porphyromonas gingivalis traffics into endoplasmic reticulum-rich-autophagosomes for successful survival in human gingival epithelial cells. Virulence. 2018;9(1):845-859.
[49] LU A, WU H. Structural mechanisms of inflammasome assembly. FEBS J. 2015;282(3):435-444.
[50] MATSUZAWA-ISHIMOTO Y, HWANG S, CADWELL K. Autophagy and Inflammation. Annu Rev Immunol. 2018;36:73-101.
[51] ARAL K, MILWARD MR, KAPILA Y, et al. Inflammasomes and their regulation in periodontal disease: A review. J Periodontal Res. 2020; 55(4):473-487.
[52] MEMMERT S, NOGUEIRA AVB, DAMANAKI A, et al. Damage-regulated autophagy modulator 1 in oral inflammation and infection. Clin Oral Investig. 2018;22(8):2933-2941.
[53] VAN DER BURGH R, NIJHUIS L, PERVOLARAKI K, et al. Defects in mitochondrial clearance predispose human monocytes to interleukin-1β hypersecretion. J Biol Chem. 2014;289(8):5000-5012.
[54] PALASKAR JN, JOSHI N, SHAH PM, et al. Influence of different implant placement techniques to improve primary implant stability in low-density bone: A systematic review. J Indian Prosthodont Soc. 2020; 20(1):11-16.
[55] JILKA RL, O’BRIEN CA. The Role of Osteocytes in Age-Related Bone Loss. Curr Osteoporos Rep. 2016;14(1):16-25.
[56] SHAPIRO IM, LAYFIELD R, LOTZ M, et al. Boning up on autophagy: the role of autophagy in skeletal biology. Autophagy. 2014;10(1):7-19.
[57] GREENHILL C. Bone: Autophagy regulates bone growth in mice. Nat Rev Endocrinol. 2016;12(1):4.
[58] CHEN K, YANG YH, JIANG SD, et al. Decreased activity of osteocyte autophagy with aging may contribute to the bone loss in senile population. Histochem Cell Biol. 2014;142(3):285-295.
[59] LIN J, XU R, SHEN X, et al. Metformin promotes the osseointegration of titanium implants under osteoporotic conditions by regulating BMSCs autophagy, and osteogenic differentiation. Biochem Biophys Res Commun. 2020;531(2):228-235.
[60] XU R, SHI G, XU L, et al. Simvastatin improves oral implant osseointegration via enhanced autophagy and osteogenesis of BMSCs and inhibited osteoclast activity. J Tissue Eng Regen Med. 2018;12(5): 1209-1219.
[61] CHEN Y, GUAN M, REN R, et al. Improved Immunoregulation of Ultra-Low-Dose Silver Nanoparticle-Loaded TiO(2) Nanotubes via M2 Macrophage Polarization by Regulating GLUT1 and Autophagy. Int J Nanomedicine. 2020;15:2011-2026.
[62] XU L, ZHANG L, WANG Z, et al. Melatonin Suppresses Estrogen Deficiency-Induced Osteoporosis and Promotes Osteoblastogenesis by Inactivating the NLRP3 Inflammasome. Calcif Tissue Int. 2018;103(4): 400-410.
[63] SALMINEN A, KAARNIRANTA K, KAUPPINEN A. Inflammaging: disturbed interplay between autophagy and inflammasomes. Aging (Albany NY). 2012;4(3):166-175.
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