[1] SHARIF K, SHARIF A, JUMAH F, et al. Rheumatoid arthritis in review: clinical, anatomical, cellular and molecular points of view. Clin Anat. 2018;31(2):216-223.
[2] 陈劲松,王中汉,常非,等.多种特殊状态下关节软骨缺损修复的组织工程技术[J].中国组织工程研究,2020,24(8):1272-1279.
[3] 张勇,温蕾.中医对风湿性关节炎的病因病机认识及治疗现状研究[J].中医临床研究,2018,10(35):144-146.
[4] 但文超,何庆勇,曲艺,等.基于网络药理学的枳术丸调治血脂异常的分子机制研究[J].世界科学技术-中医药现代化, 2019,21(11):2396-2405.
[5] 危亦林(元).世医得效方[M].北京:中国中医药出版社,1996:136.
[6] 朱震亨(元).丹溪心法[M].沈阳:辽宁科学技术出版社,1997:72.
[7] 张云程.二妙散加味治疗痹症[J].四川中医,1983(2):35.
[8] 谢莉,黄志鹏.二妙散合宣痹汤加减治疗类风湿关节炎(活动期)临床疗效观察[J].心理月刊,2020,15(14):200.
[9] 王泽,高玉亭,李振,等.二妙散对CIA大鼠关节NF-κBp50和ERK1/2表达特性的影响[J].中国免疫学杂志,2020,36(16):1951-1956.
[10] 董培良,李慧,韩华.中药网络药理学的应用与思考[J].中国实验方剂学杂志, 2020,26(17):204-211.
[11] 贺红安,王晓,陈雅桐,等.基于网络药理学和分子对接探讨升降散治疗儿童慢性扁桃体炎的作用机制[J/OL].中国中药杂志: 1-10. https://kns.cnki.net/kcms/detail/detail.
[12] ZHOU Y, ZHOU B, PACHE L, et al. Metascape provides a biologist-oriented resource for the analysis of systems-level datasets. Nat Commun. 2019;10(1):1523.
[13] VELLA D, MARINI S, VITALI F, et al. MTGO: PPI network analysis via topological and functional module identification . Sci Rep. 2018;8(1):5499.
[14] SMOLEN JS, ALETAHA D, MCINNES IB. Rheumatoid arthritis. Lancet. 2016;388 (10055):2023-2038.
[15] SCOTT DL, WOLFE F, HUIZINGA TW. Rheumatoid arthritis. Lancet. 2010;376(9746): 1094-1108.
[16] ALLARD-CHAMARD H, BOIRE G. Serologic diagnosis of rheumatoid arthritis. Clin Lab Med. 2019;39(4):525-537.
[17] GIANNINI D, ANTONUCCI M, PETRELLI F, et al. One year in review 2020: pathogenesis of rheumatoid arthritis. Clin Exp Rheumatol. 2020;38(3):387-397.
[18] 赵璐, 何东仪. 类风湿关节炎病因病机及辨证论治[J].风湿病与关节炎, 2016,5(7):71-74.
[19] 林昌松,李楠,姜玉宝,等.类风湿关节炎的中西医结合治疗研究进展[J].中华中医药杂志,2017,32(11):5020-5023.
[20] KIM HR, KIM BM, WON JY, et al. Quercetin, a plant polyphenol, has potential for the prevention of bone destruction in rheumatoid arthritis. J Med Food. 2019;22(2):152-161.
[21] HALEAGRAHARA N, MIRANDA-HERNANDEZ S, ALIM MA, et al. Therapeutic effect of quercetin in collagen-induced arthritis. Biomed Pharmacother. 2017;90:38-46.
[22] SACKS D, BAXTER B, CAMPBELL BCV, et al. Multisociety consensus quality improvement revised consensus statement for endovascular therapy of acute ischemic stroke. Int J Stroke. 2018;13(6):612-632.
[23] HUANG Y, GUO L, CHITTI R, et al. Wogonin ameliorate complete Freund’s adjuvant induced rheumatoid arthritis via targeting NF-κB/MAPK signaling pathway. Biofactors. 2020;46(2):283-291.
[24] LIU R, HAO D, XU W, et al. β-Sitosterol modulates macrophage polarization and attenuates rheumatoid inflammation in mice. Pharm Biol. 2019;57(1):161-168.
[25] DA FONSECA LJS, NUNES-SOUZA V, GOULART MOF, et al. Oxidative stress in rheumatoid arthritis: what the future might hold regarding novel biomarkers and add-on therapies. Oxid Med Cell Longev. 2019;2019:7536805.
[26] WEI Q, LV F, ZHANG H, et al. MicroRNA-101-3p inhibits fibroblast-like synoviocyte proliferation and inflammation in rheumatoid arthritis by targeting PTGS2. Biosci Rep. 2020;40(1):BSR20191136.
[27] HANNEMANN N, CAO S, ERIKSSON D, et al. Transcription factor Fra-1 targets arginase-1 to enhance macrophage-mediated inflammation in arthritis. J Clin Invest. 2019;129(7):2669-2684.
[28] SABIR JSM, EL OMRI A, BANAGANAPALLI B, et al. Dissecting the role of NF-κb protein family and its regulators in rheumatoid arthritis using weighted gene co-expression network. Front Genet. 2019;10:1163.
[29] PRASAD K. AGE-RAGE stress play a role in aortic aneurysm: a comprehensive review and novel potential therapeutic target. Rev Cardiovasc Med. 2019;20(4):201-208.
[30] 郭锦晨,刘健,万磊,等.氧化应激及炎症反应在类风湿关节炎中的研究进展[J].风湿病与关节炎,2016,5(9):68-71, 76.
[31] NOORT AR, TAK PP, TAS SW. Non-canonical NF-κB signaling in rheumatoid arthritis: Dr Jekyll and Mr Hyde. Arthritis Res Ther. 2015;17(1):15.
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