中国组织工程研究

中国组织工程研究 ›› 2011, Vol. 15 ›› Issue (46): 8730-8733.doi: 10.3969/j.issn.1673-8225.2011.46.043

• 组织构建学术探讨 tissue construction academic discussion • 上一篇    下一篇

骨骼肌的运动适应机制

张晓峰1，石  松2，魏永旺2   

  1. 1河北司法警官职业学院，河北省石家庄市  050081
    2河北传媒学院艺术体育系，河北省石家庄市  050000
  • 收稿日期:2011-05-08 修回日期:2011-09-08 出版日期:2011-11-12 发布日期:2011-11-12
  • 作者简介:张晓峰★，男，1977年生，河北省卢龙县人，汉族，2001年河北体育学院毕业，硕士，讲师，主要从事运动生理学方面的研究。 zhangxiaofeng778170@126.com

Mechanism of adaptation to exercise in skeletal muscle

Zhang Xiao-feng1, Shi Song2, Wei Yong-wang2   

  1. 1Hebei Vocational College of Judicial Police, Shijiazhuang 050081, Hebei Province, China
    2Department of Sports and Art, Hebei Institute of Communication, Shijiazhuang 050000, Hebei Province, China
  • Received:2011-05-08 Revised:2011-09-08 Online:2011-11-12 Published:2011-11-12
  • About author:Zhang Xiao-feng★, Master, Lecturer, Hebei Vocational College of Judicial Police, Shijiazhuang 050081, Hebei Province, China zhangxiaofeng778170@126.com

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409

被引次数

3

摘要：

背景：骨骼肌运动适应机制的研究对提高运动成绩，预防和治疗一些代谢紊乱性疾病具有重要意义。
目的：探讨骨骼肌运动适应的机制。
方法：应用计算机检索PubMed数据库和中文期刊全文数据库2011-03前发表的相关文章，检索词分别为“skeletal muscle, exercise, adaptation, cytoskeleton, dystrophin”和“骨骼肌，运动，适应，骨架蛋白，肌营养不良蛋白”，共检索到56篇文献，纳入所述内容与骨骼肌运动适应机制相关的文献，排除重复性研究，保留31篇进行综述。
结果与结论：激烈的运动使肌肉结构和细胞代谢产生应激反应，包括肌肉损伤和氧化应激反应。高强度的离心运动可造成肌肉超微结构损伤，但运动性肌损伤后存在肌肉再重建反应。运动训练可促进健康的个体对一氧化氮系统产生各种各样的适应，通过各种机制增强骨骼肌的生物学有效性，这些适应性变化可有效增加运动能力，对心血管系统具有保护作用。目前，大多数人类骨骼肌运动适应机制还没有被发现。

关键词: 骨骼肌, 运动, 适应, 骨架蛋白, 肌营养不良蛋白

Abstract:

BACKGROUND: Study on the mechanism of adaptation to exercise in skeletal muscle has great significance for the improvement of sports performance, the prevention and treatment of some metabolic disorders.
OBJECTIVE: To discuss the mechanism of adaptation to exercise in skeletal muscle.
METHODS: An online search of PubMed database and CNKI was performed for relevant articles published before March 2011, using key words of “skeletal muscle, exercise, adaptation, cytoskeleton, dystrophin” in English and Chinese, respectively. A total of 56 articles were collected. Articles on the mechanism of adaptation to exercise in skeletal muscle were included. A number of 31 articles were retained after excluding the repetitive ones.
RESULTS AND CONCLUSION: Intense exercise can cause stress reaction in muscle structure and cell metabolism, such as muscle damage and oxidative stress. High-strength eccentric activities may lead to the damage of muscle ultrastructure, but the muscle shows a remodeling response after exercise-induced damage. Exercise training can promote various adaptations of healthy individuals to nitric oxide system. These adaptations can somehow enhance the biological availability of skeletal muscle, consequently increase the exercise capacity and provide protection to the cardiovascular system. At present, the mechanism of adaptation to exercise in human skeletal muscle is still unclear.

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