[1] BARFIELD WR, HOLMES RE, HARTSOCK LA. Heterotopic Ossification in Trauma. Orthop Clin North Am. 2017;48:35-46.
[2] DEY D, WHEATLEY BM, CHOLOK D, et al. The traumatic bone: trauma-induced heterotopic ossification. Transl Res. 2017;186:95-111.
[3] RANGANATHAN K, LODER S, AGARWAL S, et al. Heterotopic Ossification: Basic-Science Principles and Clinical Correlates. J Bone Joint Surg Am. 2015;97:1101-1111.
[4] HWANG C, MARINI S, HUBER AK, et al. Mesenchymal VEGFA induces aberrant differentiation in heterotopic ossification. Bone Res. 2019;7:36.
[5] WANG X, LI F, XIE L, et al. Inhibition of overactive TGF-beta attenuates progression of heterotopic ossification in mice. Nat Commun. 2018;9: 551.
[6] MATSUO K, CHAVEZ RD, BARRUET E, et al. Inflammation in Fibrodysplasia Ossificans Progressiva and Other Forms of Heterotopic Ossification. Curr Osteoporos Rep. 2019;17:387-394.
[7] PETERSON JR, DE LA ROSA S, SUN H, et al. Burn injury enhances bone formation in heterotopic ossification model. Ann Surg. 2014;259: 993-998.
[8] HSU GC, MARINI S, NEGRI S, et al. Endogenous CCN family member WISP1 inhibits trauma-induced heterotopic ossification. JCI Insight. 2020;5:e135432.
[9] LODER SJ, AGARWAL S, CHUNG MT, et al. Characterizing the Circulating Cell Populations in Traumatic Heterotopic Ossification. Am J Pathol. 2018;188:2464-2473.
[10] PETERSON JR, AGARWAL S, BROWNLEY RC, et al. Direct Mouse Trauma/Burn Model of Heterotopic Ossification. J Vis Exp. 2015;102:e52880.
[11] BARTLETT CS, RAPUANO BE, LORICH DG, et al. Early changes in prostaglandins precede bone formation in a rabbit model of heterotopic ossification. Bone. 2006;38:322-332.
[12] GRUBER R, WEICH HA, DULLIN C, et al. Ectopic bone formation after implantation of a slow release system of polylactic acid and rhBMP-2. Clin Oral Implants Res. 2009;20:24-30.
[13] LIU X, KANG H, SHAHNAZARI M, et al. A novel mouse model of trauma induced heterotopic ossification. J Bone Miner Res. 2014;32:183-188.
[14] SCHNEIDER DJ, MOULTON MJ, SINGAPURI K, et al. The Frank Stinchfield Award. Inhibition of heterotopic ossification with radiation therapy in an animal model. Clin Orthop Relat Res. 1998;(355):35-46.
[15] STRONG AL, SPREADBOROUGH PJ, PAGANI CA, et al. Small molecule inhibition of non-canonical (TAK1-mediated) BMP signaling results in reduced chondrogenic ossification and heterotopic ossification in a rat model of blast-associated combat-related lower limb trauma. Bone. 2020;139:115517.
[16] WEI Z, GUO S, WANG H, et al. Comparative proteomic analysis identifies differentially expressed proteins and reveals potential mechanisms of traumatic heterotopic ossification progression. J Orthop Translat. 2022; 34:42-59.
[17] KAN L, KESSLER JA. Animal models of typical heterotopic ossification. J Biomed Biotechnol. 2011;2011:309287.
[18] XU Y, HUANG M, HE W, et al. Heterotopic Ossification: Clinical Features, Basic Researches, and Mechanical Stimulations. Front Cell Dev Biol. 2022;10:770931.
[19] BUCK RC. Regeneration of tendon. J Pathol Bacteriol. 1953; 66(1):1-18.
[20] MCCLURE J. The effect of diphosphonates on heterotopic ossification in regenerating Achilles tendon of the mouse. J Pathol. 1983;139: 419-430.
[21] LIN L, SHEN Q, XUE T, et al. Heterotopic ossification induced by Achilles tenotomy via endochondral bone formation: expression of bone and cartilage related genes. Bone. 2010;46:425-431.
[22] HUANG Y, WANG X, ZHOU D, et al. Macrophages in heterotopic ossification: from mechanisms to therapy. NPJ Regen Med. 2021;6:70.
[23] WANG H, LINDBORG C, LOUNEV V, et al. Cellular Hypoxia Promotes Heterotopic Ossification by Amplifying BMP Signaling. J Bone Miner Res. 2016;31:1652-1665.
[24] KRAFT CT, AGARWAL S, RANGANATHAN K, et al. Trauma-induced heterotopic bone formation and the role of the immune system: A review. J Trauma Acute Care Surg. 2016;80:156-165.
[25] MEDICI D, OLSEN BR. The role of endothelial-mesenchymal transition in heterotopic ossification. J Bone Miner Res. 2012;27:1619-1622.
[26] ZHANG C, ZHANG Y, ZHONG B, et al. SMAD7 prevents heterotopic ossification in a rat Achilles tendon injury model via regulation of endothelial-mesenchymal transition. FEBS J. 2016;283:1275-1285.
[27] NIEDERBICHLER AD, WESTFALL MV, SU GL, et al. Cardiomyocyte function after burn injury and lipopolysaccharide exposure: single-cell contraction analysis and cytokine secretion profile. Shock. 2006;25: 176-183.
[28] HOESEL LM, MATTAR AF, ARBABI S, et al. Local wound p38 MAPK inhibition attenuates burn-induced cardiac dysfunction. Surgery. 2009; 146(4):775-785.
[29] CHEN HC, YANG JY, CHUANG SS, et al. Heterotopic ossification in burns: our experience and literature reviews. Burns. 2009;35:857-862.
[30] HU X, SUN Z, LI F, et al. Burn-induced heterotopic ossification from incidence to therapy: key signaling pathways underlying ectopic bone formation. Cell Mol Biol Lett. 2021;26:34.
[31] FINNERTY CC, JESCHKE MG, HERNDON DN, et al. Temporal cytokine profiles in severely burned patients: a comparison of adults and children. Mol Med. 2008;14:553-560.
[32] RANGANATHAN K, PETERSON J, AGARWAL S, et al. Role of gender in burn-induced heterotopic ossification and mesenchymal cell osteogenic differentiation. Plast Reconstr Surg. 2015;135:1631-1641.
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