[1] TAIRA TM, LIMA V, PRADO DS, et al. NLRP12 Attenuates Inflammatory Bone Loss in Experimental Apical Periodontitis. J Dent Res. 2019;98(4):476-484.
[2] DIEGUES LL, COLOMBO ROBAZZA CR, Costa Hanemann JA, et al. Correlation between clinical and histopathological diagnoses in periapical inflammatory lesions. J Investig Clin Dent. 2011;2(3): 184-186.
[3] BRAZ-SILVA PH, BERGAMINI ML, MARDEGAN AP, et al. Inflammatory profile of chronic apical periodontitis: a literature review. Acta Odontol Scand. 2019;77(3):173-180.
[4] FIGUEIREDO JAP, MACHADO AM, OLIVEIRA VP, et al. Dendritic cells and their relation to apical peridontitis. Braz Oral Res. 2018; 32(suppl 1):e71.
[5] DONG M, YU X, CHEN W, et al. Osteopontin Promotes Bone Destruction in Periapical Periodontitis by Activating the NF-κB Pathway. Cell Physiol Biochem. 2018;49(3):884-898.
[6] YANG NY, ZHOU Y, ZHAO HY, et al. Increased interleukin 1α and interleukin 1β expression is involved in the progression of periapical lesions in primary teeth. BMC Oral Health. 2018;18(1): 124.
[7] RAGGATT LJ, PARTRIDGE NC. Cellular and molecular mechanisms of bone remodeling. J Biol Chem. 2010;285(33): 25103-25108.
[8] TAN X, HUANG D, ZHOU W, et al. Dickkopf-1 may regulate bone coupling by attenuating wnt/β-catenin signaling in chronic apical periodontitis. Arch Oral Biol. 2018;86:94-100.
[9] PUTKER F, BOS MP, TOMMASSEN J. Transport of lipopolysaccharide to the Gram-negative bacterial cell surface. FEMS Microbiol Rev. 2015;39(6):985-1002.
[10] YANG J, ZHAO Y, SHAO F. Non-canonical activation of inflammatory caspases by cytosolic LPS in innate immunity. Curr Opin Immunol. 2015;32:78-83.
[11] SUN Y, SHANG D. Inhibitory Effects of Antimicrobial Peptides on Lipopolysaccharide-Induced Inflammation. Mediators Inflamm. 2015;2015:167572.
[12] WANG X, SONG S, HU Z, et al. Activation of Epac alleviates inflammation and vascular leakage in LPS-induced acute murine lung injury. Biomed Pharmacother. 2017;96:1127-1136.
[13] KATOH M. Functional and cancer genomics of ASXL family members. Br J Cancer. 2013;109(2):299-306.
[14] ABDEL-WAHAB O, DEY A. The ASXL-BAP1 axis: new factors in myelopoiesis, cancer and epigenetics. Leukemia. 2013;27(1):10-15.
[15] CARBUCCIA N, MURATI A, TROUPLIN V, et al. Mutations of ASXL1 gene in myeloproliferative neoplasms. Leukemia. 2009; 23(11):2183-2186.
[16] WANG M, YANG C, ZHANG L, et al. Molecular Mutations and Their Cooccurrences in Cytogenetically Normal Acute Myeloid Leukemia. Stem Cells Int. 2017;2017:6962379.
[17] VALENCIA-MARTINEZ A, SANNA A, MASALA E, et al. Mutated ASXL1 and number of somatic mutations as possible indicators of progression to chronic myelomonocytic leukemia of myelodysplastic syndromes with single or multilineage dysplasia. Haematologica. 2017;102(9):e332-e335.
[18] ZHANG P, XING C, RHODES SD, et al. Loss of Asxl1 alters self-renewal and cell fate of bone marrow stromal cell, leading to bohring-opitz-like syndrome in mice. Stem Cell Reports. 2016;6(6): 914-925.
[19] 郭健民,章岚.自噬对成骨细胞调控作用的研究进展[J].中国骨质疏松杂志,2018,24(11):1535-1540.
[20] CAPULLI M, PAONE R, RUCCI N. Osteoblast and osteocyte: games without frontiers. Arch Biochem Biophys. 2014;561:3-12.
[21] CHEN H, LI J, WANG Q. Associations between bone-alkaline phosphatase and bone mineral density in adults with and without diabetes. Medicine (Baltimore). 2018;97(17):e0432.
[22] KOMORI T. Roles of Runx2 in Skeletal Development. Adv Exp Med Biol. 2017;962:83-93.
[23] BUSTOS F, SEPÚLVEDA H, PRIETO CP, et al. Runt-Related Transcription Factor 2 Induction During Differentiation of Wharton's Jelly Mesenchymal Stem Cells to Osteoblasts Is Regulated by Jumonji AT-Rich Interactive Domain 1B Histone Demethylase. Stem Cells. 2017;35(12):2430-2441.
[24] HAN Y, ZHANG L, XING Y, et al. Autophagy relieves the function inhibition and apoptosis‑promoting effects on osteoblast induced by glucocorticoid. Int J Mol Med. 2018;41(2):800-808.
[25] YE L, FAN Z, YU B, et al. Histone demethylases KDM4B and KDM6B promotes osteogenic differentiation of human MSCs. Cell Stem Cell. 2012;11(1):50-61.
[26] MÉNDEZ-FERRER S, MICHURINA TV, FERRARO F, et al. Mesenchymal and haematopoietic stem cells form a unique bone marrow niche. Nature. 2010;466(7308):829-834.
[27] LEE HL, YU B, DENG P, et al. Transforming Growth Factor-β- Induced KDM4B Promotes Chondrogenic Differentiation of Human Mesenchymal Stem Cells. Stem Cells. 2016;34(3):711-719.
[28] ITO K, SUDA T. Metabolic requirements for the maintenance of self- renewing stem cells. Nat Rev Mol Cell Biol. 2014;15(4): 243-256.
[29] KAWAI M, ROSEN CJ. PPARγ: a circadian transcription factor in adipogenesis and osteogenesis. Nat Rev Endocrinol. 2010;6(11): 629-636.
[30] WANG J, LI Z, HE Y, et al. Loss of Asxl1 leads to myelodysplastic syndrome-like disease in mice. Blood. 2014;123(4):541-553.
[31] MAGINI P, DELLA MONICA M, UZIELLI ML, et al. Two novel patients with Bohring-Opitz syndrome caused by de novo ASXL1 mutations. Am J Med Genet A. 2012;158A(4):917-921.
[32] ZHANG P, CHEN Z, LI R, et al. Loss of ASXL1 in the bone marrow niche dysregulates hematopoietic stem and progenitor cell fates. Cell Discov. 2018;4:4.
[33] BEDOUKIAN E, COPENHEAVER D, BALE S, et al. Bohring-Opitz syndrome caused by an ASXL1 mutation inherited from a germline mosaic mother. Am J Med Genet A. 2018;176(5): 1249-1252.
[34] URREIZTI R, GÜRSOY S, CASTILLA-VALLMANYA L, et al. The ASXL1 mutation p.Gly646Trpfs*12 found in a Turkish boy with Bohring-Opitz Syndrome. Clin Case Rep. 2018;6(8):1452-1456.
[35] LIU Y, HUANG L, HAO B, et al. Use of an Osteoblast Overload Damage Model to Probe the Effect of Icariin on the Proliferation, Differentiation and Mineralization of MC3T3-E1 Cells through the Wnt/β-Catenin Signalling Pathway. Cell Physiol Biochem. 2017; 41(4): 1605-1615.
[36] LU Y, ZHENG Q, LU W, et al. Compressive mechanical stress may activate IKK-NF-κB through proinflammatory cytokines in MC3T3-E1 cells. Biotechnol Lett. 2015;37(9):1729-1735.
[37] DING J, GHALI O, LENCEL P, et al. TNF-alpha and IL-1beta inhibit RUNX2 and collagen expression but increase alkaline phosphatase activity and mineralization in human mesenchymal stem cells. Life Sci. 2009;84(15-16):499-504.
[38] LEE HL, YI T, WOO KM, et al. Msx2 mediates the inhibitory action of TNF-alpha on osteoblast differentiation. Exp Mol Med. 2010; 42(6): 437-445.
[39] DIARRA D, STOLINA M, POLZER K, et al. Dickkopf-1 is a master regulator of joint remodeling. Nat Med. 2007;13(2):156-163.
[40] SCHETT G, DAYER JM, MANGER B. Interleukin-1 function and role in rheumatic disease. Nat Rev Rheumatol. 2016;12(1):14-24.
[41] DOMINGO-FERNÁNDEZ R, COLL RC, KEARNEY J, et al. The intracellular chloride channel proteins CLIC1 and CLIC4 induce IL-1β transcription and activate the NLRP3 inflammasome .J Biol Chem. 2017;292(29):12077-12087. |